Posts Tagged ‘intoxication’

What is a bacterial erysipelas?

Saturday, January 9th, 2010

What is a erysipelas?

Erysipelas – an infectious disease with the general intoxication of the organism and the inflammatory skin lesions. Pathogen – erysipelatous streptococcus, is stable outside the human body is very tolerant of desiccation and low temperature, dies when heated to 56 degrees Celsius for 30 minutes. The source of the disease is the patient and carrier. Contagious (contagious) is insignificant. The disease is recorded as separate cases. Infection occurs mainly in violation of the integrity of the skin with contaminated objects, tools or hands.

By the nature of lesions are distinguished:
1. erythematous form in the form of redness and swelling of the skin;
2. hemorrhagic form with the phenomena of permeability of blood vessels and bleeding;
3. bullous form of blisters on inflamed skin filled with serous fluid.

Bacterial Erysipelas

Bacterial Erysipelas

By the degree of intoxication emit – light, moderate, severe. On multiplicity – primary, recurrent, re.
As the prevalence of local manifestations – localized (nose, face, head, back, and so on.) Wandering (moving from one place to another), and metastatic.

Symptoms (signs) of erysipelas.

The incubation period of 3 to 5 days. Acute onset, sudden. In the first day more pronounced symptoms of intoxication (severe headache, chills, general weakness, possible nausea, vomiting, fever up to 39-40 degrees Celsius).

Erythematous form. After 6-12 hours of onset, there is a burning sensation, pain Expander nature of the skin – redness (erythema) and swelling at the site of inflammation. Struck erysipelas plot clearly separated from the healthy sublime sharply painful roller. The skin in the source area hot to the touch, tense. If there Punctulata hemorrhage, then talk about erythematous-hemorrhagic form of erysipelas. When bullosa erysipelas on the background of erythema at different periods after its occurrence bullous form elements – the bubbles contain a light and transparent liquid. Later, they fall off, forming a thick brown crust, rejected in 2-3 weeks. In place of bubbles can be formed and the erosion of trophic ulcers. All forms of faces accompanied the defeat of the lymphatic system – lymphadenitis, limfangitom.

Primary erysipelas often localized on the face, recurrent – in the lower extremities. There are early relapses (up to 6 months) and late (over 6 months). Contribute to the development of their associated diseases. The most important are chronic inflammatory foci of the disease lymphatic and blood vessels of the lower extremities (phlebitis, thrombophlebitis, varicose veins), diseases with a pronounced component of allergic diseases (bronchial asthma, allergic rhinitis), skin diseases (mycoses, peripheral ulcers). Relapses occur and as a result of adverse occupational factors.

Duration of the disease, the local manifestations of erythematous erysipelas are to 5.8 day of illness, in other forms may hold more than 10-14 days. Vestiges faces – pigmentation, peeling, pastoznost skin, the presence of dry dense crusts on the site of bullous elements. Perhaps lymphostasis development, leading to elephantiasis of the limbs.

Treatment of erysipelas

Depends on the form of the disease, its multiplicity, the degree of intoxication, presence of complications. Etiotropic therapy: antibiotics, penicillin in the number of average daily dose (penicillin, tetracycline, erythromycin, or oleandomitsin, oletetrin, Capoten, Hytrin, Lopid, Mevacor). Less effective drugs sulfonamides, combined chemotherapy (Bactrim, septin, Biseptol). Course of treatment is usually 8-10 days. With frequent recurrences of persistent recommend tseporin, oxacillin, and methicillin ampitsillip. It is desirable to conduct two courses of antibiotics with the change agents (the intervals between courses of 7-10 days). If often recurrent erysipelas use corticosteroids in a daily dosage of 30 mg. With persistent infiltration of displaying non-steroidal anti-inflammatory drugs – hlotazol, butadion, reopirin, etc. It is advisable the appointment of ascorbic acid, rutin, vitamin B supplements give good results autohaemotherapy. (more…)

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Epidemic parotitis (mumps) symptoms

Wednesday, December 30th, 2009

What is an epidemic parotitis (mumps)?

Mumps epidemic (mumps) – is a viral disease with general intoxication, an increase of one or more salivary glands, often defeat other glandular organs and nervous system. Pathogen – a virus spherical shape, having tropism for the glandular and nervous tissues. Not only is resistant to physical and chemical factors. The source of the disease is a sick man. Infection occurs through droplets, not exclude the possibility of contact transmission. The virus is found in saliva at the end of the incubation period for 3-8 days, after which virus isolation is terminated. Outbreaks are often local in nature.

Epidemic parotitis (mumps) symptoms

Epidemic parotitis (mumps) symptoms

Symptoms, signs and course of epidemic parotitis (mumps).

The incubation period is usually 15-19 days. There is a short prodromal (initial) period, when there is a weakness, malaise, muscle aches, headache, chilling, sleeping, appetite. With the development of inflammatory changes in the salivary gland shows signs of its destruction (dry mouth, pain in the ear, increasing in chewing, talking). The disease can be mild or severe illness.
Depending on this temperature may be from subfebrile digits to 40 degrees Celsius, intoxication also depends on the severity. Characteristic manifestation of the disease – the defeat of the salivary glands, usually the parotid. Iron increases, there is tenderness to palpation, which is particularly expressed in front of the ear, behind the ear lobe and in the mastoid process. Great diagnostic importance is the symptom Mursona-inflammatory reaction in ductless affected the parotid gland. The skin over the inflamed gland tense, shiny, swelling may extend to the neck. Increased cancer usually lasts 3 days, the maximum swelling is kept for 2-3 days. Against this background, can develop different, sometimes severe complications: meningitis, meningoencephalitis, orchitis, pancreatitis, labyrinthitis, arthritis, glomerulonephritis.

Treatment of epidemic parotitis (mumps).

Bed rest for 10 days. Compliance with the milk-vegetable diet, restriction of white bread, fat, crude fiber (cabbage).
When orhite appointed jockstrap, prednisolone 5-7 days according to the scheme Revatio, Phentrimine, Evening Primrose Oil.
When meningitis applied corticosteroids, spend lumbar puncture, intravenously injected 40% hexamine solution. In-developed acute pancreatitis is appointed by the liquid light diet, atropine, papaverine, cold on his stomach, vomiting – and chlorpromazine drugs that inhibit enzymes – gordoks, kontrikal trasilol. Weather favorable. (more…)

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Psittacosis in humans

Thursday, December 17th, 2009

Psittacosis in humans

Psittacosis – an acute infectious disease of the groups of influenza. Characterized by fever, general intoxication, lung damage, nervous system, increased liver and spleen. Reservoir and source of infection – domestic and wild birds. Currently ornithosis agent isolated from more than 140 species of birds. Most epidemiological significance have pets and pet bird, especially the urban pigeons. The share of occupational diseases have 2-5% of the total number of cases. Infection occurs through the air, but in 10% of patients indicated food infection. Pathogen ornithosis related to chlamydia, in the external environment remains up to 2-3 weeks. Resistant to sulfanilamides, sensitive to the antibiotics Noroxin,

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Yersiniosis (Pseudotuberculosis, Far Scarlatiniform fever)

Wednesday, November 4th, 2009

Yersiniosis (Pseudotuberculosis, Far Scarlatiniform fever)

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Diphtheria

Tuesday, November 3rd, 2009

Diphtheria

What is diphtheria?

Diphtheria (Greek – skin, film) – an acute infectious disease primarily of children with lesions of the throat, less often – the nose, eyes, etc., the formation of fibrinous plaque and general intoxication of the organism. Pathogen – Leffler coli produce toxins, which causes the main symptoms of the disease. Infection from patients and bacillicarriers through the air (by coughing, sneezing) and subjects. Ill not all positive Revatio online purchase. Most formed healthy bacteriocarrier. In recent years there has been a tendency to increase the incidence, seasonal rises occur in the fall.

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Lung abscess

Wednesday, September 16th, 2009

Lung abscess

Lung abscess – a limited purulent destructive process in the lung tissue with the presence of one or more cavities filled with pus and pronounced perifocal inflammatory infiltration.

Etiology.

The causative pathogen is often golden hemolytic staphylococcus, which in recent years, often combined with other pathogenic and opportunistic microorganisms (Escherichia, Pseudomonas aeruginosa, Klebsiella, viruses).

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